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DISSECTING ANEURYSM OF THORACIC AORTA: REAPPRAISAL OF RADIOLOGIC DIAGNOSIS

YACOV ITZCHAK M.D., TALMA ROSENTHAL M.D., RAPHAEL ADAR M.D., ZALLMAN J. RUBINSTEIN M.D., YAIR LIEBERMAN M.D., and VICTOR DEUTSCH M.D.

In a series of 24 cases of acute dissecting aneurysm of the aorta (not including Marfan’s disease) the diagnosis was usually suspected on the basis of the clinical picture and plain chest roentgenograms.

The most consistent clinical sign was severe pain. Absent pulses and a neurological deficit were each noted in only five patients. In many cases there was no correlation between the clinical picture and the type or the extent of the dissection.

Widening of the aortic arch and obliteration of the aortic knob with displacement of the trachea to the right are the most common signs in plain chest roentgenograms. A barium swallow examination in these cases reveals an elongated compression and displacement of the esophagus by the aortic arch. Calcification in the area of the aortic arch is the exception rather than the rule in dissecting aneurysms.

Angiography is essential for the definitive diagnosis of dissecting aneurysms. The diagnosis is based on the demonstration of two channels, either by the presence of a linear radiolucency separating the two lumens, or by differences in flow that present as delayed opacification or delayed washout.

If only the true lumen is opacified, widening of the outer extraluminal border of the aorta or narrowing of the lumen indicates the presence of a dissection. Abnormal catheter recoil and position were helpful in only two cases, and are not informative when the false lumen is catheterized. Failure to visualize main aortic branches was not always due to involvement by the dissection. It can also be caused by reduced flow due to severe proximal compression of the main lumen.

The exact location of the intimal tears is usually not demonstrated unless additional injections are made in the area assumed to contain the tear.

If only the false lumen is opacified in the ascending aorta, this can be recognized by the demonstration of a blind end, by failure to visualize the sinuses of Valsalva, from flattening of the medial border of the opacified channel, and from delayed washout in the blind end.


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M. H. Drucker, L. A. Woods, and S. M. Austin
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J. Schneiderman, R. Walden, and R. Adar
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Vascular and Endovascular Surgery, March 1, 1977; 11(2): 52 - 54.
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