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MR Findings in Cardiac Amyloidosis

Rohan I. vanden Driesen, Richard E. Slaughter and Wendy E. Strugnell

Cardiovascular MR Research Centre, Prince Charles Hospital, Rode Rd., Chermside, Brisbane, QLD Q4032, Australia.


Figure 1
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  		Fig. 1 —Four-chamber steady-state free precession image of 64-year-old man with cardiac amyloidosis shows diffuse thickening of myocardium and mild atrial enlargement.

 

Figure 2
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  		Fig. 2A —61-year-old man with cardiac amyloidosis. Short-axis views from postgadolinium delayed enhancement images show widespread enhancement.

 

Figure 3
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  		Fig. 2B —61-year-old man with cardiac amyloidosis. two-chamber long-axis views from postgadolinium delayed enhancement images show widespread enhancement.

 

Figure 4
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  		Fig. 3A —62-year-old woman with cardiac amyloidosis. Short-axis views from postgadolinium delayed enhancement images show widespread enhancement.

 

Figure 5
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  		Fig. 3B —62-year-old woman with cardiac amyloidosis. and two-chamber long-axis views from postgadolinium delayed enhancement images show widespread enhancement.

 

Figure 6
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  		Fig. 4A —72-year-old man with cardiac amyloidosis. Short-axis views obtained before gadolinium administration confirm that observed myocardial signal change represents true delayed myocardial enhancement.

 

Figure 7
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  		Fig. 4B —72-year-old man with cardiac amyloidosis. and after gadolinium administration confirm that observed myocardial signal change represents true delayed myocardial enhancement.

 

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