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Gadobenate Dimeglumine-Enhanced Liver MRI as the Sole Preoperative Imaging Technique: A Prospective Study of Living Liver Donors

Su Kyung An1, Jeong Min Lee1, Kyung-Suk Suh2, Nam Jun Lee2, Se Hyung Kim1, Young Joon Kim1, Joon Koo Han1 and Buyng Ihn Choi1

1 Department of Radiology and Institute of Radiation Medicine, Seoul National University College of Medicine, 28, Yongon-dong, Chongno-gu, Seoul 110-744, South Korea.
2 Department of General Surgery, Seoul National University College of Medicine, Seoul, South Korea.


Figure 1
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Fig. 1A 25-year-old man with anomaly of right posterior duct draining to left hepatic duct. Coronal thick-slab turbo spin-echo T2-weighted MR cholangiography (A) and 3D T1-weighted MR cholangiography with gadobenate dimeglumine enhancement (B) show anomaly of right posterior duct draining into left hepatic duct.

 

Figure 2
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Fig. 1B 25-year-old man with anomaly of right posterior duct draining to left hepatic duct. Coronal thick-slab turbo spin-echo T2-weighted MR cholangiography (A) and 3D T1-weighted MR cholangiography with gadobenate dimeglumine enhancement (B) show anomaly of right posterior duct draining into left hepatic duct.

 

Figure 3
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Fig. 1C 25-year-old man with anomaly of right posterior duct draining to left hepatic duct. Intraoperative contrast-enhanced cholangiography confirms this anomaly.

 

Figure 4
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Fig. 2A 23-year-old man with biliary trifurcation. Coronal thick-slab T2-weighted MR cholangiography (A) and 3D T1-weighted MR cholangiography with gadobenate dimeglumine enhancement (B) show anomaly of trifurcation of bile duct at conjugation level.

 

Figure 5
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Fig. 2B 23-year-old man with biliary trifurcation. Coronal thick-slab T2-weighted MR cholangiography (A) and 3D T1-weighted MR cholangiography with gadobenate dimeglumine enhancement (B) show anomaly of trifurcation of bile duct at conjugation level.

 

Figure 6
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Fig. 2C 23-year-old man with biliary trifurcation. Intraoperative contrast-enhanced cholangiography confirms this anomaly.

 

Figure 7
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Fig. 3 33-year-old woman with replaced right hepatic artery. Coronal maximum-intensity-projection image obtained from arterial phase 3D MR angiography with gadobenate dimeglumine enhancement shows anomaly of right hepatic artery arising from superior mesenteric artery (arrow).

 

Figure 8
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Fig. 4 22-year-old man with portal vein trifurcation. Coronal maximum-intensity-projection image obtained from portal venous phase 3D MR angiography with gadobenate dimeglumine enhancement shows anomaly of trifurcation of right anterior portal vein, right posterior portal vein, and left portal vein.

 

Figure 9
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Fig. 5 43-year-old woman with right inferior accessory hepatic vein. Coronal maximum-intensity-projection image obtained from hepatic venous phase 3D MR angiography with gadobenate dimeglumine enhancement shows anomaly of right inferior accessory hepatic vein (arrow) draining directly into inferior vena cava.

 

Figure 10
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Fig. 6A 16-year-old boy with uneven fatty infiltration of liver. In-phase (A) and opposed-phase (B) T1-weighted gradient-echo images show signal drop in liver, suggesting fatty liver with focal sparing in segment IV and segment I.

 

Figure 11
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Fig. 6B 16-year-old boy with uneven fatty infiltration of liver. In-phase (A) and opposed-phase (B) T1-weighted gradient-echo images show signal drop in liver, suggesting fatty liver with focal sparing in segment IV and segment I.

 

Figure 12
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Fig. 7A 22-year-old man with peribiliary cysts. Minimum-intensity-projection image of MDCT shows lobulating, contoured structures with low attenuation along portal vein; this finding can be dilated bile ducts or peribiliary cystic lesions.

 

Figure 13
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Fig. 7B 22-year-old man with peribiliary cysts. Coronal thick-slab T2-weighted MR cholangiography shows multiple round structures with high signal intensity. CT and T2-weighted MR cholangiography cannot differentiate peribiliary cysts from choledochal cysts.

 

Figure 14
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Fig. 7C 22-year-old man with peribiliary cysts. Three-dimensional T1-weighted MR cholangiography with gadobenate dimeglumine enhancement shows normal bile duct; therefore, cystic structures are peribiliary cysts. Segmental nonvisualization of common hepatic duct was due to compression by one of the peribiliary cysts (arrow).

 

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