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MRI of Hepatic Adenomatosis: Initial Observations with Gadoxetic Acid Contrast Agent in Three Patients

Olivier Giovanoli1,2, Markus Heim3, Luigi Terracciano4, Georg Bongartz1 and Hans P. Ledermann5

1 Department of Radiology, University Hospital Basel, Basel, Switzerland.
2 Present address: Institute of Radiology, Kantonsspital Aarau, Tellstrasse, 5001 Aarau, Switzerland.
3 Department of Gastroenterology, University Hospital Basel, Basel, Switzerland.
4 Department of Pathology, University Hospital Basel, Basel, Switzerland.
5 Imamed Radiologie Nordwest, Basel, Switzerland.


Figure 1
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  		Fig. 1A —27-year-old woman with histologically proven liver adenomatosis and underlying glycogen storage disease type Ib. Lesions are barely visible on unenhanced T1-weighted image (A) and hyperintense on T2-weighted image (B).

 

Figure 2
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  		Fig. 1B —27-year-old woman with histologically proven liver adenomatosis and underlying glycogen storage disease type Ib. Lesions are barely visible on unenhanced T1-weighted image (A) and hyperintense on T2-weighted image (B).

 

Figure 3
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  		Fig. 1C —27-year-old woman with histologically proven liver adenomatosis and underlying glycogen storage disease type Ib. On 20-minute delayed image after gadoxetic acid administration, all lesions are strongly hypointense in relation to strong gadoxetic acid uptake by surrounding normal liver parenchyma.

 

Figure 4
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  		Fig. 2A —41-year-old woman with histologically proven liver adenomatosis. In dynamic, gadoxetic acid–enhanced images after 25 seconds (A), 70 seconds (B), 3 minutes (C), and 15 minutes (D), all lesions show strong arterial enhancement (A). Enhancing regions are mostly isointense to liver parenchyma in venous imaging (B). In delayed phases C and D, lesions are hypointense compared with gadoxetic acid enhancement of surrounding normal liver parenchyma. Two lesions with exceptional peripheral contrast enhancement are shown in this slice.

 

Figure 5
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  		Fig. 2B —41-year-old woman with histologically proven liver adenomatosis. In dynamic, gadoxetic acid–enhanced images after 25 seconds (A), 70 seconds (B), 3 minutes (C), and 15 minutes (D), all lesions show strong arterial enhancement (A). Enhancing regions are mostly isointense to liver parenchyma in venous imaging (B). In delayed phases C and D, lesions are hypointense compared with gadoxetic acid enhancement of surrounding normal liver parenchyma. Two lesions with exceptional peripheral contrast enhancement are shown in this slice.

 

Figure 6
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  		Fig. 2C —41-year-old woman with histologically proven liver adenomatosis. In dynamic, gadoxetic acid–enhanced images after 25 seconds (A), 70 seconds (B), 3 minutes (C), and 15 minutes (D), all lesions show strong arterial enhancement (A). Enhancing regions are mostly isointense to liver parenchyma in venous imaging (B). In delayed phases C and D, lesions are hypointense compared with gadoxetic acid enhancement of surrounding normal liver parenchyma. Two lesions with exceptional peripheral contrast enhancement are shown in this slice.

 

Figure 7
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  		Fig. 2D —41-year-old woman with histologically proven liver adenomatosis. In dynamic, gadoxetic acid–enhanced images after 25 seconds (A), 70 seconds (B), 3 minutes (C), and 15 minutes (D), all lesions show strong arterial enhancement (A). Enhancing regions are mostly isointense to liver parenchyma in venous imaging (B). In delayed phases C and D, lesions are hypointense compared with gadoxetic acid enhancement of surrounding normal liver parenchyma. Two lesions with exceptional peripheral contrast enhancement are shown in this slice.

 

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